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H 2 O 2 -induced autophagic responses were attenuated when ERK1/2 activity were suppressed by their inhibitor U0126, suggesting that ERK activation are required for autophagy induced by H 2 O 2 in HUVECs, implying that an ERK1/2-HDAC6 pathway is at least partially involved in H 2 O 2 -mediated autophagy
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We measured several intermediate and end-point biochemical markers to assess the validity of our working hypothesis that carnitine, acting via an endothelial-mediated blood flow regulatory mechanism, could reduce catabolism of purines, free radical formation, sarcolemma disruption, and perceived soreness
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