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Acta Med Okayama
485 These findings indicate that mitochondria iron overload, combined with ROS formation and subsequent ferroptosis, intensifies the cardiotoxic effects of DOX-induced cardiomyopathy and may be an important mechanism
cell proliferation and differentiation: MAPKK (MEK) 1/2 [S221/S225], ERK1 [T185/Y187], ERK2 [T185/Y187], ERK1/2 [T202/Y204]
Onset generally occurs in late adolescence or early adulthood
Research has examined roles in tissue remodelling, anti-inflammatory pathways, antioxidant activity and gene expression related to collagen synthesis
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