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discovered that oestrogen-activated GPER in CAFs promotes the expression of GLUL and lactate dehydrogenase B (LDHB), enhances the survival of Triple Negative Breast Cancer(TNBC) cells both in vitro and ex vivo, and increases chemotherapy resistance [130]
(PubMed) Vermeiren S, Vella-Azzopardi R, Beckwee D, et al
E.WolffS.et al (2016)
In addition to this attack, which might be caused by homocysteinylation of the vascular wall proteins by homocysteine and/or its oxidized products (homocysteic acid, homocysteine sulfinic acid, and homocysteine thiolactone), in vitro and in vivo work suggests that reduced homocysteine can inhibit endothelial cell turn-over (Wang et al, 1997) and promote smooth muscle cell proliferation (Lubec et al, 1996b
The recent approval of empagliflozin for children and adolescents with T2D marks a crucial step forward, with initial studies reporting promising results
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