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Estrogen has been shown to upregulate endogenous MOTS-c expression in preclinical models, which may partly explain why metabolic deterioration accelerates in perimenopause when estrogen falls precipitously [2]

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In this Critical Perspective, we discuss a central limitation of current Model-Informed Precision Dosing in critically ill patients: many dosing decisions are still based on intermittent snapshots of a physiology that is continuously changing

The lack of regulatory oversight raises concerns about the consistency and reliability of compounded medications